Washington, D.C. 20549


                                    FORM 8-K

                                 CURRENT REPORT

                     PURSUANT TO SECTION 13 or 15(d) OF THE

                         SECURITIES EXCHANGE ACT OF 1934


         Date of report (Date of earliest event reported) July 16, 2002

                                   ALTEON INC.
               (Exact Name of Registrant as Specified in Charter)

        Delaware                     001-16043                   13-3304550
 (State or Other Juris-             (Commission               (I.R.S. Employer
diction of Incorporation)           File Number)             Identification No.)

 170 Williams Drive, Ramsey, New Jersey                             07446
(Address of Principal Executive Offices)                          (Zip Code)

        Registrant's telephone number, including area code (201) 934-5000

          (Former Name or Former Address, If Changed Since Last Report)

Item 5. Other Events

      On July 16, 2002, Alteon Inc. issued the following press release:


- Phase IIa DIAMOND Trial Focused on Diastolic Dysfunction, a Major Cause of
Congestive Heart Failure -

RAMSEY, N.J., Jul 16, 2002 /PRNewswire-FirstCall via COMTEX/ -- Alteon Inc.
(Amex: ALT) announced today that it has begun a Phase IIa trial of its lead
A.G.E. Crosslink Breaker, ALT-711, in patients with diastolic heart failure
(DHF). DHF is a condition characterized by the inability of the heart to relax
properly and fill with blood, due to stiffening of the heart and impaired
relaxation of the left ventricle. Diastolic dysfunction is estimated to account
for 30-50% of all heart failure cases, which total nearly 5 million in the U.S.

The Phase IIa study, called the DIAMOND [Distensibility Improvement with ALT-711
Remodeling in Diastolic Heart Failure] trial, will seek to determine if ALT-711
can have a direct remodeling impact on the stiff heart, thus offering promise as
a novel therapy for a medical condition that is currently poorly treated. In
previous human clinical testing, ALT-711 has shown the ability to restore
elasticity to blood vessel walls by cleaving pathological glucose-protein
structures called Advanced Glycosylation End-product (A.G.E.) Crosslinks.(1)
Additionally, in several preclinical studies, including a study that was
presented at the September 2001 meeting of the American Heart Association's
Council for High Blood Pressure Research,(2) ALT-711 has been shown to normalize
the thickening of the left ventricle and remodel the heart.

ALT-711 is currently in three additional human clinical trials. Two Phase IIb
trials, the SAPPHIRE and SILVER trials, are actively enrolling over 630 patients
in order to evaluate the compound's effectiveness in patients with elevated
systolic blood pressure [systolic hypertension] without or with enlargement of
the left ventricle of the heart [left ventricular hypertrophy or LVH]. Systolic
hypertension results from age-related or diabetes-related stiffening of the
large arteries, and is a key factor in coronary heart disease in individuals
over the age of 50. Alteon is also conducting a Phase I pharmacokinetic study in
patients with end-stage renal disease undergoing peritoneal dialysis, a patient
population with significant cardiovascular disease.


The Phase IIa pilot trial of ALT-711 is being conducted at Wake Forest
University Baptist Medical Center and the Medical University of South Carolina
in patients at least 60 years of age with isolated diastolic heart failure.
Primary endpoints will include changes in aortic stiffness, as measured by
state-of-the-art magnetic resonance imaging, and exercise tolerance. Effects on
LVH, diastolic filling and quality of life will also be assessed. Approximately
20 patients will receive 210 mg of ALT-711 twice daily on an

open-label, out-patient basis for 16 weeks. A historical control group of DHF
patients who did not receive ALT-711 will be used for comparison. Data from the
trial is anticipated during 2003.

Dalane W. Kitzman, M.D., Associate Professor of Medicine and Cardiology at Wake
Forest University Baptist Medical Center, and an investigator in the trial,
recently helped to identify diastolic heart failure as a more common condition
than previously thought, particularly in people aged 65 and older, and
especially in women. "Our study showed that the majority of older congestive
heart failure patients have diastolic dysfunction, a less understood form of
heart failure,"(3) said Dr. Kitzman. "We need more research on the progression
of the disease and how best to treat it."

"ALT-711 may provide a novel treatment approach for diastolic dysfunction," said
Michael Zile, M.D., FACC, FAHA, Charles Ezra Daniel Professor of Medicine,
Medical University of South Carolina, and an investigator in the trial. "We are
pleased to be a part of this important study."

About Alteon

Alteon is developing several new classes of drugs that reverse or slow down
diseases of aging and complications of diabetes. These compounds impact a
fundamental pathological process caused by protein-glucose complexes called
Advanced Glycosylation End-products (A.G.E.s). The formation and crosslinking of
A.G.E.s are an inevitable part of the aging and diabetic complications process,
and leads to a loss of flexibility and function in body tissues, organs and
vessels. Alteon is initially developing therapies for cardiovascular and kidney
diseases in older or diabetic individuals.

Alteon has created a library of novel classes of compounds targeting the A.G.E.
Pathway. These include A.G.E. Crosslink Breakers, A.G.E. Formation Inhibitors
and Glucose Lowering Agents. The Company's lead A.G.E. Crosslink Breaker,
ALT-711, is being evaluated in the Phase IIb SAPPHIRE clinical trial focused on
patients with systolic hypertension, and the Phase IIb SILVER trial in patients
with systolic hypertension and left ventricular hypertrophy. ALT-711 is also
undergoing Phase IIa evaluation in patients with diastolic heart failure, the
DIAMOND trial, and a Phase I investigation in end-stage renal disease patients
undergoing peritoneal dialysis, a patient population that has significant
cardiovascular disease. Other A.G.E. compounds are being evaluated for skin
aging, and additional indications. For more information on Alteon, visit the
company's web site at .

1 Kass DA, Shapiro EP, Kawaguchi M, Capriotti AR, Scuteri A, deGroof RC, Lakatta
EG. Improved arterial compliance by a novel advanced glycation end- product
crosslink breaker. Circulation 2001; 104:1464-70, Rapid Track Publication.

2 Veronesi M, Adam AG, Raij L. Abstract - ALT-711, A collagen cross- link
breaker, decreases myocardial fibrosis and improves endothelial dysfunction in
hypertensive Dahl

salt rats. American Heart Association 55th Annual Fall Conference and Scientific
Sessions of the Council for High Blood Pressure Research, September 2001.

3 Kitzman DW, Gardin JM, Gottdiener JS, Arnold A, Boineau, R, Aurigemma G,
Marino EK, Lyles M, Cushman M, Enright PL. Importance of heart failure with
preserved systolic function in patients > 65 years of age. American Journal of
Cardiology (2001; 87:413-9).

For more information on diastolic dysfunction and congestive heart failure,
please see; search for
diastolic dysfunction

Any statements contained in this press release that relate to future plans,
events or performance are forward-looking statements that involve risks and
uncertainties including, but not limited to, those relating to technology and
product development (including the possibility that early clinical trial results
may not be predictive of results that will be obtained in large-scale testing or
that any clinical trials will not demonstrate sufficient safety and efficacy to
obtain requisite approvals or will not result in marketable products),
regulatory approval processes, intellectual property rights and litigation,
competitive products, ability to obtain financing, and other risks identified in
Alteon's filings with the Securities and Exchange Commission. The information
contained in this press release is accurate as of the date indicated. Actual
results, events or performance may differ materially. Alteon undertakes no
obligation to publicly release the result of any revision to these
forward-looking statements that may be made to reflect events or circumstances
after the date hereof or to reflect the occurrence of unanticipated events

                                 * * * * * * *

      Pursuant to the requirements of the Securities Exchange Act of 1934, the
Registrant has duly caused this report to be signed on its behalf by the
undersigned hereunto duly authorized.

                                      Alteon Inc.

                                           /s/ Kenneth I. Moch
                                          Kenneth I. Moch
                                          President and Chief Executive Officer

Dated: July 18, 2002